Reply To: Covid as a veil for establishment preperation for environmental collapse


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#62715
Clark

Pooh, there are so many references I don’t have time to work out which are most relevant, but you could try some of these:

– 27. Mao L, Jin H, Wang M, et al.: Neurologic manifestations of hospitalized patients with coronavirus disease 2019 in Wuhan, China. JAMA Neurol 2020;77:1–9.

– 31. Lechien JR, Chiesa-Estomba CM, De Siati DR, et al.: Olfactory and gustatory dysfunctions as a clinical presentation of mild-to-moderate forms of the coronavirus disease (COVID-19): A multicenter European study. Eur Arch Otorhinolaryngol 2020;277:2251–2261.

– 32. Giacomelli A, Pezzati L, Conti F, et al.: Self-reported olfactory and taste disorders in SARS-CoV-2 patients: A cross-sectional study. Clin Infect Dis 2020;ciaa330.

– 33. Spinato G, Fabbris C, Polesel J, et al.: Alterations in smell or taste in mildly symptomatic outpatients with SARS-CoV-2 infection. JAMA 2020;323:2089–2090.

– 34. Beltran-Corbellini A, Chico-Garcia JL, Martinez-Poles J, et al.: Acute-onset smell and taste disorders in the context of Covid-19: A pilot multicenter PCR-based case-control study. Eur J Neurol 2020. [Epub ahead of print]; DOI: 10.1111/ene.14273.

– 35. Yan CH, Faraji F, Prajapati DP, Ostrander BT, DeConde AS: Self-reported olfactory loss associates with outpatient clinical course in Covid-19. Int Forum Allergy Rhinol 2020;10:821–831.

– 36. Moein ST, Hashemian SMR, Mansourafshar B, Khorram-Tousi A, Tabarsi P, Doty RL: Smell dysfunction: A biomarker for COVID-19. Int Forum Allergy Rhinol 2020. [Epub ahead of print]; DOI: 10.1002/alr.22587.

I found those as citations in this paper:

What HIV in the Brain Can Teach Us About SARS-CoV-2 Neurological Complications?

There is also this preprint; the effect has been demonstrated in hamsters:

COVID-19-associated olfactory dysfunction reveals SARS-CoV-2 neuroinvasion and persistence in the olfactory system

“…olfactory and taste dysfunction are rather common in COVID-19, especially in pauci-symptomatic patients which constitutes the most frequent clinical manifestation of the infection. We conducted a virologic, molecular, and cellular study of the olfactory system from COVID-19 patients presenting acute loss of smell, and report evidence that the olfactory epithelium represents a highly significant infection site where multiple cell types, including olfactory sensory neurons, support cells and immune cells, are infected. Viral replication in the olfactory epithelium is associated with local inflammation. Furthermore, we show that SARS-CoV-2 induces acute anosmia and ageusia in golden Syrian hamsters, both lasting as long as the virus remains in the olfactory epithelium and the olfactory bulb. Finally, olfactory mucosa sampling in COVID-19 patients presenting with persistent loss of smell reveals the presence of virus transcripts and of SARS-CoV-2-infected cells, together with protracted inflammation. Viral persistence in the olfactory epithelium therefore provides a potential mechanism for prolonged or relapsing symptoms of COVID-19, such as loss of smell, which should be considered for optimal medical management and future therapeutic strategies.”